Dos años más tarde presentó episodios recurrentes de taquicardia a lat/min no revertió con verapamilo i.v. Tras la cardioversión eléctrica de la taquicardia, Diagnosis and cure of Wolff-Parkinson-White or paroxysmal supraventricular. Request PDF on ResearchGate | Actualización en taquicardia ventricular | La Una taquicardia mal tolerada requiere cardioversión eléctrica, mientras que una . El registro de la tira de ritmo (tras amiodarona intravenosa) corrobora un diagnóstico de taquicardia ventricular. 4. La cardioversión eléctrica resulta efectiva.

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A QRS axis that is deviated to the right superior quadrant has long been recognized as being caused by VT, and this phenomenon is similar to an R wave in lead aVR.

Key clinical characteristics of inherited long QT syndrome LQTS are shown, including prolongation of QT interval on electrocardiogram ECGcommonly associated arrhythmia torsades de pointesclinical manifestation, and long-term outcomes. Note the baseline Txquicardia prolongation, with abrupt lengthening of the QT interval after the pause, followed by the onset of polymorphic ventricular tachycardia, which suddenly terminates.

Puede existir y no ser obvia en ECG. This tachycardia arises more anteriorly close to the interventricular septum. Stable — This refers to a patient showing no evidence of hemodynamic compromise despite a sustained rapid heart rate.

When any supgaventricular criteria 1 to 3 was present, VT was diagnosed; when absent, the next criterion was analyzed.

The rationale for these criteria is eminently reasonable. In this setting, emergent synchronized cardioversion is the treatment of choice regardless of the mechanism of the arrhythmia.


Negative concordancy is diagnostic for a Electricq arising in the apical area of the heart fig One to one ventriculo-atrial conduction during VT.

The rhythm is more likely originating in ventricular tissue. The QRS complexes are not preceded by P waves.


See “Unstable patient” below. The frontal QRS axis shows left axis deviation. The insertion of the accessory pathway in the free wall of the right ventricle results in sequential right to left ventricular activation and a wide QRS complex. If they are P waves, they occur in 1: When the onset of the arrhythmia is available for analysis, a period of irregularity “warm-up phenomenon”suggests VT.

Si no se sincroniza: Eur Heart J ; Regularity — VT is generally regular, although slight variation in the RR intervals is taquicardix seen. Ablation of supraventricular tachycardia resistant to medical treatment and electrical cardioversion in a pregnant woman.

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Also the presence of AV conduction disturbances during sinus rhythm make it very unlikely that a broad QRS tachycardia in that patient has a supraventricular origin and, as already shown in fig 11, a QRS width during tachycardia more narrow that during sinus rhythm points to a VT. See “Overview of advanced elecrrica life support in adults” and see “Overview of basic cardiovascular life support in adults”.

It may occur in AV junctional tachycardia with BBB after cardiac surgery or during digitalis intoxication. If all precordial leads are predominantly positive, the differential diagnosis is an antidromic tachycardia using a left sided accessory pathway or a Supraventricula.

When in doubt, do not give verapamil or adenosine; procainamide should be used instead. On the left sinus rhythm is present with a ellectrica wide QRS because of anterolateral myocardial infarction and pronounced delay in left ventricular activation. They are often amenable to cure by radiofrequency ablation.



The origin of the QRS rhythm may be in the AV junction, with associated intraventricular aberration, or in fascicular or ventricular tissue. See “General principles of the implantable cardioverter-defibrillator”.

Idiopathic outflow tract tachycardias are usually exertion or stress related arrhythmias. How to cite this article. These notches might be P waves, or part of the QRS complexes themselves. It is of interest that a QRS width of more than 0. Nondiagnostic J point elevation in precordial leads V1 and V2. It is important to recognise this pattern because this site of origin of the VT cannot be treated with catheter ablation in contrast to the tachycardias depicted in panel A and B C, Eje QRS: The arrhythmia is often responsive to treatment with b blockers, sotalol9 or calcium channel blockers and can also be amenable to transcatheter ablation.

It arises on or near to the septum near the left posterior fascicle. The QRST complexes of the sinus-conducted beats are normal. As described in the text, lead V1 during LBBB clearly shows signs pointing to a supraventricular origin of the tachycardia.